For what reason excess belly fat is associated with a higher risk of cardiovascular disease. 

Due to its metabolic and physiological effects, abdominal fat, especially visceral fat, increases heart disease risk. Below are some reasons why belly obesity increases heart disease risk:

Activated visceral fat releases adipokines, which are inflammatory. These pro-inflammatory adipokines increase inflammation. Cardiovascular disorders are linked to chronic inflammation.

Abdominal obesity is significantly linked to insulin resistance, which reduces cell response to insulin. Insulin resistance raises blood sugar and increases the risk of type 2 diabetes and heart disease.

Visceral fat releases free fatty acids into the circulation. High levels of circulating free fatty acids can harm lipid metabolism and cause atherosclerosis, which narrows arteries and increases the risk of heart attacks and strokes.

Hormonal imbalances: Abdominal obesity increases cortisol, a stress hormone, and decreases adiponectin, an anti-inflammatory hormone. Hormonal alterations might cause metabolic and cardiovascular issues.

Excess abdominal obesity is commonly associated with high blood pressure, aberrant lipid profiles (elevated triglycerides, low HDL cholesterol), and prothrombosis.

Reducing cardiovascular risk requires lifestyle changes like nutrition, exercise, and weight management to address abdominal obesity. 

Additionally, managing risk factors like blood pressure and cholesterol is crucial to heart health. Maintaining cardiovascular health requires specialized counsel from healthcare providers.

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